To determine whether eAGR2 acts through an autocrine/paracrine mechanism, conditioned media from HBEC cells stably infected with bare vector (HBEC-EV) or overexpressing AGR2 (HBEC-AGR2) were added to HBEC organoids (Number 6L)

To determine whether eAGR2 acts through an autocrine/paracrine mechanism, conditioned media from HBEC cells stably infected with bare vector (HBEC-EV) or overexpressing AGR2 (HBEC-AGR2) were added to HBEC organoids (Number 6L). ER-retention website (KTEL), and is sufficient, by itself, to