Infante-Duarte (Experimental and Clinical Study Center, a joint assistance between the Charit-Universit?tsmedizin Berlin and the Max-Delbrck Center for Molecular Medicine, Berlin, Germany). and NeuN during mESC differentiation in vitro as well as during murine mind development and probe model of
We further found that BTG3 overexpression could inhibited the mTOR pathway and AMPK activation, and BTG3 silence showed the opposite effects
We further found that BTG3 overexpression could inhibited the mTOR pathway and AMPK activation, and BTG3 silence showed the opposite effects. In the end, we also studied Rabbit Polyclonal to FST the effects of hypoxia and/or propofol on BTG3 expression
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